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The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
Proglucagon mRNA expression in NCI-H716 cells. Cells were incubated... | Download Scientific Diagram
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FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
NCI-H716 cells as a model for endocrine differentiation in colorectal cancer
NCI-H716 cells as a model for endocrine differentiation in colorectal cancer
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Peptide production and secretion in GLUTag, NCI-H716, and STC-1 cells: a comparison to native L-cells in: Journal of Molecular Endocrinology Volume 56 Issue 3 (2016)
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
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The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
Immunofluorescent staining of NCI-H716 cells with antiGLP-1 antibody.... | Download Scientific Diagram
CGA immunoreactivity in NCI-H716 cells in varous conditions. A:... | Download Scientific Diagram